Benefits

• Supports normal cellular energy production
• Helps maintain healthy body composition
• Supports brain and cognitive health
• May help promote mitochondrial density and function with age

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What’s Inside (Ingredient Highlights)

• Coenzyme Q10 – Crucial for ATP (energy) production and antioxidant regeneration.
• R-Lipoic Acid – Supports enzyme function and regenerates antioxidants.
• L-Carnitine – Transports fats into mitochondria for energy production.
• D-Ribose – Essential for ATP formation and energy coenzyme synthesis.
• Malic Acid + Succinic Acid – Core players in the Krebs cycle, vital for cellular energy.
• Activated B-Vitamins – Fuel for energy metabolism and mitochondrial function.
• Resveratrol + Curcumin – Powerful plant antioxidants to support mitochondrial integrity.

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How to Use

Take 4 capsules per day with meals or as directed by your health-care practitioner. (Divided dosing recommended.)

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What to Avoid

Consult with your health-care practitioner about your specific circumstances and any questions you may have about this product.

*These statements have not been evaluated by the Food and Drug Administration. This product is not intended to diagnose, treat, cure, or prevent any disease.

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Purity & Safety

• No gluten, dairy, soy, or GMOs
• Non-habit forming
• Free from artificial ingredients and preservatives
• Manufactured using high-quality, research-backed ingredients
• Always third-party tested for purity and potency

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Ingredient List

Thiamine (Thiamin HCL), Riboflavin (Riboflavin-5-Phosphate), Niacin (Niacinamide), Vitamin B6 (Pyridoxal-5-Phosphate), Vitamin B12 (MecobalActive®), Manganese (TRAACS® Manganese Glycinate Chelate), L-Carnitine, D-Ribose, Malic Acid, Succinic Acid, Coenzyme Q10, R-Lipoic Acid, Trans-Resveratrol (Veri-te™), Curcuminoid Powder (Curcuma longa rhizomes), Pantethine (Pantesin®), cellulose capsule, microcrystalline cellulose, dicalcium phosphate, silicon dioxide, vegetable stearate.

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Ingredient Partners

• TRAACS® – Albion Laboratories, Inc.
• MecobalActive® – HEALTHTECH BIO ACTIVES, S.L.U.
• Pantesin® – Kyowa Pharma Chemical Co., Ltd.
• Curcumin C3 Complex® – Sabinsa Corporation (U.S. Patent No. 5,861,415)
• Veri-te™ – Evolva or its affiliates

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Research:

1. Rose J, Brian C, Woods J, et al. Mitochondrial dysfunction in glial cells: implications for neuronal homeostasis and survival. Toxicology. 2017;391:109‑115. doi:10.1016/j.tox.2017.06.011.
2. Tilokani L, Nagashima S, Paupe V, Prudent J. Mitochondrial dynamics: overview of molecular mechanisms. Essays Biochem. 2018;62(3):341‑360. doi:10.1042/EBC20170104.
3. Rossignol DA, Frye RE. Mitochondrial dysfunction in autism spectrum disorders: a systematic review and meta‑analysis. Mol Psychiatry. 2012;17(3):290‑314. doi:10.1038/mp.2010.136.
4. Briston T, Hicks AR. Mitochondrial dysfunction and neurodegenerative proteinopathies: mechanisms and prospects for therapeutic intervention. Biochem Soc Trans. 2018;46(4):829‑842. doi:10.1042/BST20180025.
5. van Horssen J, van Schaik P, Witte M. Inflammation and mitochondrial dysfunction: a vicious circle in neurodegenerative disorders? Neurosci Lett. 2019;710:132931. doi:10.1016/j.neulet.2017.06.050.
6. Elfawy HA, Das B. Crosstalk between mitochondrial dysfunction, oxidative stress, and age related neurodegenerative disease: etiologies and therapeutic strategies. Life Sci. 2019;218:165‑184. doi:10.1016/j.lfs.2018.12.029.
7. Subramaniam SR, Chesselet MF. Mitochondrial dysfunction and oxidative stress in Parkinson’s disease. Prog Neurobiol. 2013;106‑107:17‑32. doi:10.1016/j.pneurobio.2013.04.004.
8. Park JS, Davis RL, Sue CM. Mitochondrial dysfunction in Parkinson’s disease: new mechanistic insights and therapeutic perspectives. Curr Neurol Neurosci Rep. 2018;18(5):21. doi:10.1007/s11910‑018‑0829‑3.
9. Bansal Y, Kuhad A. Mitochondrial dysfunction in depression. Curr Neuropharmacol. 2016;14(6):610‑618.
doi:10.2174/1570159×14666160229114755.
10. Chistiakov DA, Shkurat TP, Melnichenko AA, Grechko AV, Orekhov AN. The role of mitochondrial dysfunction in cardiovascular disease: a brief review. Ann Med. 2018;50(2):121‑127. doi:10.1080/07853890.2017.1417631
11. Sánchez‑Domínguez B, Bullón P, Román‑Malo L, et al. Oxidative stress, mitochondrial dysfunction and, inflammation common events in skin of patients with fibromyalgia. Mitochondrion. 2015;21:69‑75. doi:10.1016/j.mito.2015.01.010.
12. Morris G, Maes M. Mitochondrial dysfunctions in myalgic encephalomyelitis/chronic fatigue syndrome explained
by activated immuno‑inflammatory, oxidative and nitrosative stress pathways. Metab Brain Dis. 2014;29(1):19‑36. doi:10.1007/s11011‑013‑9435‑x.
13. Zhang J, Bao Y, Zhou X, Zheng L. Polycystic ovary syndrome and mitochondrial dysfunction. Reprod Biol Endocrinol. 2019;17(1):67. doi:10.1186/s12958‑019‑0509‑4.
14. Bhatti JS, Bhatti GK, Reddy PH. Mitochondrial dysfunction and oxidative stress in metabolic disorders ‑ A step towards mitochondria based therapeutic strategies. Biochim Biophys Acta Mol Basis Dis. 2017;1863(5):1066‑1077. doi:10.1016/j. bbadis.2016.11.010.
15. Wei PZ, Szeto CC. Mitochondrial dysfunction in diabetic kidney disease. Clin Chim Acta. 2019;496:108‑116. doi:10.1016/j.cca.2019.07.005.